Monilinia Or Brown Rot
Monilia Laxa, Monilia Fructigena
Pathogen:
Type:
Fungus
Risk:
HIGH
Cerezo
Monilia
WHO CAUSES IT?
Monilia laxa and Monilia fructigena are phytopathogenic fungi responsible for the disease known as monilinia or brown rot, mainly affecting cherry crops and other stone fruit trees. Both pathogens produce asexual spores known as conidia, which are spread primarily by wind and rainwater. During periods of high humidity, conidia germinate on the surface of flowers, fruits or wounds on the plant, penetrating the tissues and causing infections. Fungi can survive in the form of mycelium in the remains of mummified plants and fruits that remain in the soil or on the tree during the winter. In spring, when environmental conditions are favorable, conidia are released and new infections begin in plant tissues. Additionally, spores can directly infect wounds caused by insects or human activity, facilitating the spread of the disease.
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SYMPTOMS
Monilinia or brown rot significantly affects the cherry tree, causing serious damage to both flowers and fruits, which reduces the production and quality of the harvest. Infection can begin in flowers during spring and spread to fruits as they ripen, causing rapid decay and death of infected tissues.
- Appearance of brown Taches on flowers and fruits
- Withering and death of flowers and young shoots
- Development of concentric rings of gray spores in infected areas
- Mummification of affected fruits, which remain on the tree
- Discoloration and necrosis of branches and shoots
- Premature fall of affected fruits
TEMPERATURE AND HUMIDITY
15°C - 25°C
70% - 90%
TRANSMISSION ROUTES
Wind-borne conidia, Rain, Contaminated pruning tools, Infected plant material, Insects
Do you want to remove this disease? Choose how you want to treat it.
TREATMENTS
Chemical treatments
• SULFUR 70% [SC] P/V
• SULFUR 72% [SC] P/V
• CAPTAN 47.5% [SC] P/V
• CAPTAIN 80% [WG] P/P
• CIPRODINIL 37.5% + FLUDIOXONIL 25% [WG] P/P
• Cyprodinil 50% [WG] P/P
• DIFENOCONAZOLE 1.67% [EC] P/V
• DIFENOCONAZOLE 25% [EC] P/V
• PHENHEXAMIDE 50% [SC] P/V
• PHENHEXAMIDE 50% [WG] P/P
• PHENPIRAZAMINE 50% [WG] P/P
• FLUDIOXONIL 50% [WG] P/P
• FLUOPYRAM 20% + TEBUCONAZOLE 20% [SC] P/V
• FLUOPYRAM 50% [SC] P/V
• POTASSIUM HYDROGEN CARBONATE 0.425% [AL] P/V
• POTASSIUM HYDROGEN CARBONATE 85% [SP] P/P
• CUPRIC HYDROXIDE 13.6% (EXPR. IN CU) + COPPER OXYCHLORIDE 13.6% (EXPR. IN CU) [SC] P/V
• CUPRIC HYDROXIDE 20% (EXPR. IN CU) [WG] P/P
• CUPRIC HYDROXIDE 25% (EXPR. IN CU) [WG] P/P
• CUPRIC HYDROXIDE 30% (EXPR. IN CU) [WG] P/P
• CUPRIC HYDROXIDE 35% (EXPR. IN CU) [WG] P/P
• CUPRIC HYDROXIDE 36% (EXPR. IN CU) [SC] P/V
• CUPRIC HYDROXIDE 40% (EXPR. IN CU) [WG] P/P
• CUPRIC HYDROXIDE 50% (EXPR. IN CU) [WP] P/P
• ISOFETAMID 40% [SC] P/V
• MANDESTROBIN 25% [SC] P/V
• MEFENTRIFLUCONAZOLE 7.5% [SC] P/V
• COPPER OXYCHLORIDE 30% (EXPR. IN CU) [WP] P/P
• COPPER OXYCHLORIDE 35% (exp. in Cu) [WG] P/P
• COPPER OXYCHLORIDE 35% (EXPR. IN CU) [WG] P/P
• COPPER OXYCHLORIDE 37.5% (EXPR. IN CU) [WG] P/P
• COPPER OXYCHLORIDE 38% (EXPR. IN CU) [SC] P/V
• COPPER OXYCHLORIDE 50% (EXPR. IN CU) [WG] P/P
• COPPER OXYCHLORIDE 50% (EXPR. IN CU) [WP] P/P
• COPPER OXYCHLORIDE 52% (EXPR. IN CU) [SC] P/V
• COPPER OXYCHLORIDE 70% (EXPR. IN CU) [SC] P/V
• COPPER OXYCHLORIDE 70% [SC] P/V
• CUPROUS OXIDE 50% (EXPR. IN CU) [WP] P/P
• CUPROUS OXIDE 75% (EXPR. IN CU) [WG] P/P
• PIRACLOSTROBIN 6.7% + BOSCALIDE 26.7% ( ) [WG] P/P
• PIRACLOSTROBIN 6.7% + BOSCALIDE 26.7% [WG] P/P
• CALCIUM POLYSULPHIDE 38% [DC] P/V
• CUPROCALCIC SULFATE 12.4% (EXPR. IN CU) [SC] P/V
• CUPROCALCIC SULFATE 20% (EXPR. IN CU) [WG] P/P
• CUPROCALCIC SULFATE 20% (EXPR. IN CU) [WP] P/P
• CUPROCALCIC SULFATE 20% (EXPR. IN CU) [WG] P/P
• CUPROCALCIC SULFATE 20% [WP] P/P
• TRIBASIC COPPER SULFATE 40% (EXPR. IN CU) [WG] P/P
• TEBUCONAZOLE 20% [EW] P/V
• TEBUCONAZOLE 25% [EW] P/V
• TEBUCONAZOLE 25% [WG] P/P
• TEBUCONAZOLE 50% + TRIFLOXISTROBIN 25% [WG] P/P
Treatments allowed in organic farming
• SULFUR 70% [SC] P/V
• SULFUR 72% [SC] P/V
• POTASSIUM HYDROGEN CARBONATE 0.425% [AL] P/V
• POTASSIUM HYDROGEN CARBONATE 85% [SP] P/P
• CUPRIC HYDROXIDE 13.6% (EXPR. IN CU) + COPPER OXYCHLORIDE 13.6% (EXPR. IN CU) [SC] P/V
• CUPRIC HYDROXIDE 20% (EXPR. IN CU) [WG] P/P
• CUPRIC HYDROXIDE 25% (EXPR. IN CU) [WG] P/P
• CUPRIC HYDROXIDE 30% (EXPR. IN CU) [WG] P/P
• CUPRIC HYDROXIDE 35% (EXPR. IN CU) [WG] P/P
• CUPRIC HYDROXIDE 36% (EXPR. IN CU) [SC] P/V
• CUPRIC HYDROXIDE 40% (EXPR. IN CU) [WG] P/P
• CUPRIC HYDROXIDE 50% (EXPR. IN CU) [WP] P/P
• COPPER OXYCHLORIDE 30% (EXPR. IN CU) [WP] P/P
• COPPER OXYCHLORIDE 35% (exp. in Cu) [WG] P/P
• COPPER OXYCHLORIDE 35% (EXPR. IN CU) [WG] P/P
• COPPER OXYCHLORIDE 37.5% (EXPR. IN CU) [WG] P/P
• COPPER OXYCHLORIDE 38% (EXPR. IN CU) [SC] P/V
• COPPER OXYCHLORIDE 50% (EXPR. IN CU) [WG] P/P
• COPPER OXYCHLORIDE 50% (EXPR. IN CU) [WP] P/P
• COPPER OXYCHLORIDE 52% (EXPR. IN CU) [SC] P/V
• COPPER OXYCHLORIDE 70% (EXPR. IN CU) [SC] P/V
• COPPER OXYCHLORIDE 70% [SC] P/V
• CUPROUS OXIDE 50% (EXPR. IN CU) [WP] P/P
• CUPROUS OXIDE 75% (EXPR. IN CU) [WG] P/P
• CUPROCALCIC SULFATE 12.4% (EXPR. IN CU) [SC] P/V
• CUPROCALCIC SULFATE 20% (EXPR. IN CU) [WG] P/P
• CUPROCALCIC SULFATE 20% (EXPR. IN CU) [WP] P/P
• CUPROCALCIC SULFATE 20% (EXPR. IN CU) [WG] P/P
• CUPROCALCIC SULFATE 20% [WP] P/P
• TRIBASIC COPPER SULFATE 40% (EXPR. IN CU) [WG] P/P
Biological treatments
• BACILLUS AMYLOLIQUEFACIENS (strain AH2) (1x10E11 CFU/l) [SC] P/V
• BACILLUS AMYLOLIQUEFACIENS (subsp. plantarum, strain D747) 25% [WG] P/P
• BACILLUS SUBTILIS (STRAIN QST 713) 1.34% [SC] P/V
• SACCHAROMYCES CEREVISIAE (strain LAS02) 96.1% (1 x 10E13 cfu/Kg) [WG] P/P
• Trichoderma atroviride strain SC1 1(1 x 10E10 cfu)/g [WG] P/P
Recommendations
- Carry out adequate pruning to eliminate and destroy branches and mummified fruits that can serve as sources of inoculum.
- Apply specific fungicides during the critical stages of cherry tree development, especially during flowering and before harvest.
- Avoid sprinkler irrigation and reduce the density of the plantation to improve air circulation and reduce humidity in the foliage.
- Regularly inspect the orchard to detect early symptoms of the disease and quickly eliminate affected parts.
- Use varieties of cherry trees that are more resistant to Monilia laxa and Monilia fructigena.
- Implement integrated pest management (IPM) practices to control insects that can cause wounds and facilitate the entry of pathogens.
- Maintain good soil health through crop rotations and the incorporation of organic matter.
- Promote biodiversity in the garden to encourage the presence of natural enemies of pathogens.
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*The recommended treatments are recommendations based on the authorities' databases and in no way replace the guidelines established by the legislation of each country.
*Products shown are recommendations and not our own products. As Amazon Associates, we earn revenue from purchases of recommended products.
TREATMENTS
Homemade remedies
There are no home treatments
Natural allies
Chemical treatments
There are no treatments for this disease. Treatments are directed at the insect vectors that transmit it. See insect treatments.
RECOMMENDATIONS
REPELLENT PLANTS
EFFECTIVE PRODUCTS TO ELIMINATE THIS DISEASE
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*The recommended treatments are recommendations based on the authorities' databases and in no way replace the guidelines established by the legislation of each country.
*Products shown are recommendations and not our own products. As Amazon Associates, we earn revenue from purchases of recommended products.
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